Himalaya Abana
Benefits of Abana : Cardio-protective, lowers cholesterol, lowers LDL, increases HDL, lowers blood pressure
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Abana from Himalaya Herbals is a good herbal ayurvedic formulation. Abana has been formulated for most favorable heart health. It helps to lower the cholesterol and LDL while raising HDL. It works as a cardio-protective by improving the contractility of the heart and by reducing sensitivity to adrenergic stimulation. It also reduces the platelet aggregation.
Abana can be taken as a daily supplement to prevent the heart attacks, reduce the risk factors of coronary heart disease, reduce hypertension while well as nervousness and anxiety. Abana does not cause any momentous change in the blood pressure of normotensive individuals.
Benefits of Abana
- Abana is a cardio-protective
- Abana helps to get reduce hypertension
- Abana helps in blood circulation
- Abana helps to reduce the cholesterol and lower serum lipids
- Abana helps conquer nervousness and anxiety which leads to cardiac neurosis
Indications for taking Abana
- Mild to moderate hypertension
- Hyperlipidemic conditions
- As an adjuvant in the therapy of angina with the cardiac risk factors
- Cardiovascular and cerebrovascular conditions those requiring inhibition of platelet aggregation
Directions for taking Himalaya Abana
commonly 2-3 tablets, twice daily best taken with warm water. Please consult your physician to prescribe the dosage that best suits to your condition. Natural products treat not just the symptoms but the body as a whole and take time for assimilation and results.
Abana from Himalaya Herbals
Himalaya Abana is from the renowned Himalaya Herbals brand and endorsed by over 250,000 doctors worldwide and used by the customers in over 60 countries. Himalaya Herbals products have been researched clinically and consistent to guarantee bioequivalence. Bioequivalence refers to get make certain that the product on the market is so equivalent to the one on which clinical trials were efficiently conducted. Himalaya Herbal Healthcare uses the chromatographic fingerprinting, it is one of the most refined standardization techniques, to ensure dependable quality and performance
Abana ingredients and composition
- Arjuna (Terminalia arjuna) 30mg
- Ashvagandha (Withania somnifera) 20mg
- Badranj boya (Nepeta hindostana) 20mg
- Dashamoola 20mg
- Guduchi (Tinospora cordifolia) 10mg
- Amalaki (Emblica officinalis) 10mg
- Haritaki (Terminalia chebula) 10mg
- Bhringaraja (Eclipta alba Syn. E.prostrata) 10mg
- Yashti-madhu (Glycyrrhiza glabra) 10mg
- Shatavari (Asparagus racemosus) 10mg
- Punarnava (Boerhaavia diffusa) 10mg
Pdrs:
- Guggulu (Balsamodendron mukul Syn. Commiphora wightii) (Purified) 30mg
- Shilajeet (Purified) 20mg
- Mandukaparni (Centella asiatica) 10mg
- Shankhapushpi (Convolvulus pluricaulis) 10mg
- Vishnu priya (Ocimum sanctum Syn. O.tenuiflorum) 10mg
- Jatamansi (Nardostachys jatamansi) 10mg
- Pippali (Piper longum) 10mg
- Yavani (Carum copticum Syn. Trachyspermum ammi) 10mg
- Sunthi (Zingiber officinale) 10mg
- Nagapashana bhasma 10mg
- Shankh bhasma 10mg
- Makardhwaj 10mg
- Musta (Cyperus rotundus) 5mg
- Vacha (Acorus calamus) 5mg
- Vidanga (Embelia ribes) 5mg
- Lavanga (Syzygium aromaticum) 5mg
- Jyotishmati (Celastrus paniculatus) 5mg
- Chandana (Santalum album) 5mg
- Ela (Elettaria cardamomum) 5mg
- Shatapushpa (Foeniculum vulgare) 5mg
- Satapatrika (Rosa damascena Syn. R.centifolia) 5mg
- Tavak patra (Cinnamomum cassia) 5mg
- Abhrak bhasma 5mg
- Mukta pishti (Pearl pishti) 5mg
- Akik pishti (Agate pishti) 5mg
- Yeshab pishti (Vyomashma pishti) 5mg
- Yakut pishti (Manikya pishti) 5mg
- Praval pishti (Coral pishti) 5mg
- Kumkuma (Crocus sativus) 2mg
Abana Research and Clinical Studies
Double-blind comparative clinical trial of Abana and Simvastatin in Hyperlipidaemia
Venkataramaiah, H., M.D., D.M. (Cardiology), Professor of Cardiology, Jayadeva Institute of Cardiology, Jayanagar East End, Bangalore, India.
[Corresponding author: Kala Suhas Kulkarni, M.D., Medical Advisor, R&D Center, The Himalaya Drug Company, Makali, Bangalore, India]
INTRODUCTION
Observational studies have standard hyperlipidaemia as an independent risk factor for coronary artery disease. It is now proved that hyperlipidaemia is an independent risk factor for ischemic stroke. Additional evidence from the impending studies have shown the relationship between plasma cholesterol levels and risk of stroke. Reduction in the plasma cholesterol is usher by significant decrease in the incidence of coronary artery disease and stroke.
Data from the entity randomised trials and meta-analyses of randomised trials regularly show a reduction in risk for both fatal and nonfatal coronary heart disease by following primary and secondary prevention. A recent comprehensive overview that by Law and colleagues incorporated data from the 28 trials of cholesterol reduction, by including 6 multiple intervention trials that each been had a cholesterol-reducing arm. This summary indicated that almost a 10% reduction in serum cholesterol level resulted into highly significant reductions mortality from coronary heart disease. These data extracted from the randomised trials are consistent with observational data when treatment lasts that 5 years or more. A 10% reduction in cholesterol levels was really associated with a 25% reduction in coronary events that amongst persons treated for more than 5 years. These findings from the meta-analyses are also supported by the recent reports as of the Scandinavian study and the West of Scotland Coronary Prevention Study.
Herbs have been used whereas ancient times for reducing body lipids. Reports on all garlic studies performed, found cholesterol was in fact lowered by an standard of 9-12% over a one-to-four month period9. Guggul, a mixture of essence that taken from the plant Commiphora mukul, is an accepted treatment for the elevated cholesterol in India and has been a mainstay of the Ayurvedic approach in to preventing atherosclerosis. One trial studying the effects of the guggul reported that serum cholesterol dropped by 17.5%10. In a further report comparing guggul to the drug clofibrate, average fall in serum cholesterol was somewhat greater in the guggul group while HDL cholesterol rose in 60% of people responding to guggul, even as clofibrate treatment did not promote HDL. Wild yam another herb commonly used has also been reported to raise the HDL cholesterol in preliminary research12. With the above leads we actually planned a double-blind comparative clinical study using Abana and Simvastatin.
MATERIALS AND METHODS
Seventy patients were evaluated for all-purpose health and lipid profile through a medical history and a thorough physical examination. Patients with secondary hyperlipidaemia, alcoholism, or body weight more than the 15% above the ideal for their height were debarred from the study. Baseline cholesterol and triglycerides of estimation were also carried out. The patients those showing serum total cholesterol levels more than 200 mg/dL or serum triglyceride levels more than 200 mg/dL were integrated in the study. After screening fifty patients qualified for the study, their ages also ranged from 29 to 64 years, with a median of 46. There were 37 male and 13 female patients. Each patient undergo routine hematological and biochemical laboratory investigations. Patients were also asked not to eat any food, except for water, for 12 to 14 hours before taking blood samples. Routine urine analysis and electrocardiography was also conceded out. The study planned was double blind, randomized comparative study for 8 weeks. The written and informed consent was attain from every the patients. Patients took 2 capsules of the drug before breakfast and at bedtime. The patients had to visit every 2 weeks for 8 weeks. A registered dietician interviewed the patients and instructed them down to have diet with low cholesterol and saturated fats. The clinical side effects if any were recorded at every visit and discussed with the patient to know the basic nature, severity and frequency. Patients were seen by the same dietician at every clinic visit all over the study and were instructed to get follow the same diets and to maintain weight, physical activity levels and smoking frequency for the duration of the study. To evaluate diet compliance, patients made for written records of the quantity and type of food that consumed in 4 consecutive days, by including a weekend, between visits. These food diaries were kept on special forms that were then translated into the computer language and analyzed by a program designed for that purpose. Patients reported their usual physical activity and smoking habits on a particular card at every visit. All but three patients did not smoke cigarettes. Repeat laboratory investigations and electrocardiography were done after conclusion of the study.
Concomitant medications were check throughout the study. Twenty-three patients took no other drugs, 11 took asprin, acetaminophen, or both, 8 took antihistamines / antiallergic preparations, vitamins, or mineral supplements, 7 took inconsequential tranquilizers, sedatives, or laxatives, 4 took nonsteroidal anti-inflammatory drugs, 4 took antacids or anticholinergic drugs, and 2 took antibiotics. The results were analyzed by paired 't' test.
RESULTS
Of the 50 patients who entered the active-treatment segment of the study, 3 were debarred because of non-compliance. The patients also followed fairly uniform dietary patterns during the trial and their compliance was assured by routine interviews with the dietician and review of the computer's analysis of dietary records at each clinic visit. Routine follow-up by the dietician resulted in good overall dietary compliance and accounted for the attainment, in several patients of normal cholesterol levels in both the drug treatment. Results of those patients taking Abana showed a reduction of cholesterol as of 215.3 ± 7.42 mg/dl to 192.3 ± 9.08 mg/dl. Reduction in cholesterol as of 204.2 ± 8.43 to 157.0 ± 7.54 mg/dl occurred with Simvastatin. Triglycerides levels were also reduced from 216.0 ± 26.37 mg/dl to 187.7 ± 21.28 mg/dl and 214.7 ± 34.09 to 173.5 ± 23.23 mg/dl with Abana and Simvastatin respectively. In HDL, levels were increased in a analogous fashion with Abana and Simvastatin treatment. Although, the rise in HDL cholesterol was analogous in both the drugs, Simvastatin produced increase of HDL-cholesterol marginally elevated than the Abana. However in view of the risks involved in taking statin drugs, Abana is the safer alternative
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